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Atlas of Liver Pathology by Gary C. Kanel

By Gary C. Kanel

The Atlas of Liver Pathology, by way of Drs. Gary C. Kanel and Jacob Korula, offers the visible counsel you must correctly diagnose all sorts of liver disorder. prepared via sickness sort, it issues out significant histological positive aspects, updates illness parameters with new pictures and diagrams, and is helping you realize the scientific points of every disorder. It presents quickly and handy connection with nearly the entire liver issues in general visible at the present time. Nine-hundred-plus fine quality, full-color photos trap the gross and histological presentation of liver pathology excellent for comparability to the specimens you stumble upon in perform.

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B Figure 2-21 Fulminant hepatitis. A, Medium-power view of the parenchyma shows total liver cell dropout with lymphocytic infiltrates and Kupffer cell hyperplasia. Proliferating ductules are seen at the edges of the field and represent activation and proliferation of stem cells in the reparative process. B, High-power view shows the collapsed parenchyma to be composed of hyperplastic Kupffer cells, macrophages, residual apoptotic hepatocytes, and lymphocytes. because on hematoxylin and eosin (H&E) stain, the areas of collapse may resemble true fibrous bands.

The portal tract shows lymphocytes spilling out into the periportal region, surrounding individual and small groups of hepatocytes (periportal interface inflammatory activity, “piecemeal” necrosis), a morphologic indicator of active disease. Figure 2-28 Chronic viral hepatitis, active. Lymphocytes can be seen spilling out of the portal tracts at the edges of the field and surrounding individual and small group of hepatocytes (periportal interface inflammatory activity), a feature characteristic of the active stage of the disease.

Bile ductular proliferation is prominent. 26 Part I / Liver and Hepatobiliary Pathology with Clinical Correlations A Figure 2-22 Fulminant hepatitis. Residual spared hepatocytes are seen in this field (submassive hepatic necrosis). 3. Cytokeratins (particularly CK7, CK8, CK18, and CK19): Staining of the proliferating cholangioles is enhanced. B Figure 2-21 Fulminant hepatitis. A, Medium-power view of the parenchyma shows total liver cell dropout with lymphocytic infiltrates and Kupffer cell hyperplasia.

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