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Asthma in the Workplace, Third Edition by David I. Bernstein, Moira Chan-Yeung, Jean-Luc Malo, I.

By David I. Bernstein, Moira Chan-Yeung, Jean-Luc Malo, I. Leonard Bernstein

Addressing each key component to occupational asthma-including illness mechanisms, scientific prognosis, remedy, and different types of causative agents-this 3rd variation offers a professional survey of the main influential advances and examine within the box. providing new and accelerated chapters on genetics, environmental tracking, pathophysiology, pores and skin and pulmonary interactions, and the surveillance and prevention of occupational bronchial asthma, this consultant will stand on my own because the most recent resource at the subject.

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Additional info for Asthma in the Workplace, Third Edition

Sample text

The key element common to all of the aforementioned definitions is the presence of a causal relationship between workplace exposure and the development of workrelated asthma. It therefore seems logical to limit the definition of OA to those conditions in which the asthma is induced or caused by occupation, as originally proposed by professor Jack Pepys: ‘‘Having made a diagnosis of asthma (‘‘widespread airways obstruction reversible over short periods of time, either spontaneously or as a result of treatment’’), it is then necessary in occupational asthma to establish a relationship to the work as recommended by Ramazzini in 1713’’ (9).

6). Both commercial and purified preparations of 6-amino-penicillanic acid elicited quite different reactions to the ampicillin, coming on after 10 hours and maximal at 16 hours (Fig. 7). The difference between the two materials is the addition of phenylglycine acid chloride as a side chain to the 6-amino-pencillanic acid to make ampicillin. Identical asthmatic reactions were given after oral administration of a dose of the relevant antibiotic (Fig. 8). Immediate-type reactions to phenylglycine being made in large amounts have also been reported (61).

The first polyurethane foams were made during World War II by I. G. Farben in Germany, using TDI as an activator. 2 million tons are likely to be used in the United States in the next 10 years involving many thousands of workers. Occupational-type tests were made with epoxy resin systems containing phthalic acid anhydride, TMA, and triethylene tetramine (64). Their sensitivity could be shown by the test consisting of as little as a single breath of fume from heated materials. Their diagnostic value was shown in a patient thought to have asthma due to TDI to which he, however, gave a negative reaction; an acute immediate reaction was given to a heated cured resin containing phthalic acid anhydride.

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